...and his name was Weston A. Price MS., D.D.S., F.A.G.D.
He was a dentist, so he got to see inside a lot of mouths. What he saw worried him - a lot. There's no point in me copying and pasting stuff that someone else has written, so read all about it at http://www.westonaprice.org/Weston-A.-Price-DDS.html
To sum-up. Mr Price travelled around the world with his wife looking inside the mouths of relatively primitive people, compared to the residents of Cleveland. What he saw impressed him - a lot. Considering that primitive people don't have access to clean water, food, medicine and other modern aids to living, they had hardly any tooth decay, gum disease or overcrowded teeth.
So he took lots of photographs, made lots of measurements and asked lots of questions. When he finished his travels, he wrote a book called Nutrition and Physical Degeneration A Comparison of Primitive and Modern Diets and Their Effects. You can read it at http://www.journeytoforever.org/farm_library/price/pricetoc.html
It's worth your while reading this book. The conclusions:-
Don't eat food that's been "buggered about with". Eat as much natural food as you can and prepare it in a way that maximises the nutrients and minimises the anti-nutrients. Don't worry about the fat in it, as fat contains the fat-soluble Vitamins A, D, E & K (but not enough D unless you live on a diet of oily fish, seals & beluga whales). For more information, see http://www.westonaprice.org/
That is all.
Friday, January 30, 2009
Thursday, January 29, 2009
So, when & where did it all go wrong?
No, I'm not talking about Demand Five! I'm talking about us, modern man & woman. We have improved hygiene, clean water & food, modern medicine, anti-biotics, anti-virals etc. We should be enjoying good health and vitality into our nineties. We're not, though. Degenerative diseases like Type 2 Diabetes, Coronary Heart Disease, Cancer, Dementia, IBS etc are afflicting increasing numbers of people (including youngsters) and are even starting to reduce our longevity statistics. Why?
On one side of the fence are the anti-animal fat brigade who claim that animal fats are the cause of all our health problems and that we should all be eating more vegetable fats and reducing our cholesterol.
On the other side of the fence are the anti-carb brigade who claim that carbohydrates are the cause of all our health problems and that we should all be eating less carbohydrates and increasing our fat consumption.
I'm sure you can guess where I am. I have the splinters to prove it!
In 1911, hydrogenated vegetable oil (Crisco) entered the marketplace. So, in 1911, fat turned bad! See The rise and fall of Crisco.
Interestingly, rates of Coronary Heart Disease started to rise from 1920, 9 years later. Co-incidence?
Our genes may have not changed much in the last few hundred thousand years, but our lifestyles certainly have. We now live mostly sedentary lives (which makes our muscles less sensitive to insulin). We now live and work mostly indoors (which makes us deficient in Vitamin D).
We now don't eat much oily fish. Our vegetables contain much less omega-3 fat than they used to (to make them stay fresh for longer). Our meat now contains much more omega-6 and much less omega-3 fat than it used to (due to feeding animals on grains). These changes make us deficient in omega-3 fat (which makes our muscles less sensitive to insulin).
We now eat loads of refined carbohydrate (which causes unstable blood glucose & insulin levels) and loads of processed foods (which makes us deficient in Magnesium and fibre/fiber).
As a result of all of the above changes, we have many modern diseases. We can't blame it on one factor only. So, what can we do? Here's one suggestion....
Good health!
On one side of the fence are the anti-animal fat brigade who claim that animal fats are the cause of all our health problems and that we should all be eating more vegetable fats and reducing our cholesterol.
On the other side of the fence are the anti-carb brigade who claim that carbohydrates are the cause of all our health problems and that we should all be eating less carbohydrates and increasing our fat consumption.
I'm sure you can guess where I am. I have the splinters to prove it!
In 1911, hydrogenated vegetable oil (Crisco) entered the marketplace. So, in 1911, fat turned bad! See The rise and fall of Crisco.
Interestingly, rates of Coronary Heart Disease started to rise from 1920, 9 years later. Co-incidence?
Our genes may have not changed much in the last few hundred thousand years, but our lifestyles certainly have. We now live mostly sedentary lives (which makes our muscles less sensitive to insulin). We now live and work mostly indoors (which makes us deficient in Vitamin D).
We now don't eat much oily fish. Our vegetables contain much less omega-3 fat than they used to (to make them stay fresh for longer). Our meat now contains much more omega-6 and much less omega-3 fat than it used to (due to feeding animals on grains). These changes make us deficient in omega-3 fat (which makes our muscles less sensitive to insulin).
We now eat loads of refined carbohydrate (which causes unstable blood glucose & insulin levels) and loads of processed foods (which makes us deficient in Magnesium and fibre/fiber).
As a result of all of the above changes, we have many modern diseases. We can't blame it on one factor only. So, what can we do? Here's one suggestion....
Good health!
Labels:
Cancer,
Coronary Heart Disease,
Crisco,
Dementia,
Diabetes,
Fibre,
Hydrogenated vegetable oil,
IBS,
Magnesium,
Omega-3,
Omega-6,
Processed foods,
Refined carbohydrate,
Type 2 Diabetes,
Vitamin D,
Vitamin D3
Calorie Restriction Improves Memory
This is actually one of the first things I documented when I started Calorie Restriction. It was so obvious, my ability to concentrate, to learn, my mind just seemed so clear. This was seen my test scores because back when I was eating a bad diet I couldn't concentrate or grasp difficult things, or it would take me a long time. After I went on CR I started to learn so many things and retain that information far better.
Wednesday, January 28, 2009
I do NOT believe they wanted to be doing that!
As Harry Enfield's "It's only meee!" character used to say. I'm talking about Demand Five, Channel 5 TV's on-line "watch television on demand" service.
Up until last Friday, I was watching Neighbours using Firefox 3. Yes, I know that's really sad! On Monday, I went to watch Neighbours and was greeted by a new Media Player window displaying:
"Your flash plug-in is out of date
Please download the latest version here"
No problemo, thought I. I updated my flash plug-in and went back to watch Neighbours, to be greeted by the new Media Player window displaying:
"Your flash plug-in is out of date
Please download the latest version here"
Uh-oh! I contacted the Demand Five Support Team informing them of my problem. I got the following reply:
"Greetings,
Thank you for contacting the Demand Five Support Team.
Please try to access with Internet Explorer..."
I stopped reading at that point as I don't use Internet Explorer. I e-mailed the Support Team informing them of that fact. I got the following reply:
"Greetings,
We apologize for the inconvenience, since our contents are related with DRM therefore our service is only compatible with Internet Explorer. All other browsers (e.g., Firefox, Opera, Safari, etc...) are not compatible at this time..."
They left out Google Chrome! So basically, Demand Five just alienated a large number of their users by making their site incompatible with every browser except Internet Explorer. Thanks a bunch! (That's an ironic thank you, for the benefit of foreign readers).
As I really wanted to watch Neighbours, I ran Internet Explorer, updated my flash plug-in and off I went. Monday's episode played O.K. but Tuesday's episode stopped after the 15 second Weight-Watcher's intro'. I e-mailed the Support Team informing them of that fact. I got the following reply:
"Greetings,
Please upgrade your DRM security at the following site:
http://go.microsoft.com/FWLink?LinkID=34506
The Demand Five Support Team
downloadsupport@five.tv
AM"
I clicked the link and pressed the Upgrade button. It didn't work. I e-mailed the Support Team informing them of that fact. I got the following reply:
"Greetings,
Thank you for contacting the Demand Five Support Team.
Please follow the instructions given below:
Please open Windows Media Player (WMP)
In the menu area at the top of the WMP window, click "Tools"
If "Tools" is not visible, Right-click on the upper bar area on WMP and a Menu-list will appear
In the list that appears, choose "Options"
In the window that opens, the "Player" tab will be the first tab displayed
Please ensure that both "Download codecs automatically" and "Connect to the Internet" are selected
Please select the "File Types" tab
Click "Select All", located below and to the right of the list
Click "Apply", located at the bottom of the "Options" window
Please select the "Network" tab
In the "Protocols for MMS URLs" section, un-check "RTSP/UDP" and "RTSP/TCP"
Now, re-check "RTSP/UDP" and "RTSP/TCP"
All three protocols should now be selected
Click "OK" at the bottom of the "Options" Window
Please close Windows Media Player."
It worked. I e-mailed the Support Team informing them of that fact and also asked them why Demand Five couldn't be as easy to use as BBC iPlayer. I didn't get a reply. I have posted the above information so that you too can watch Neighbours....everybody needs good Neighbours....
UPDATE: The Demand Five media player now works with Firefox 3 and Safari. This may mean that it now also works with Opera & Chrome.
According to the support page, it's still not compatible with Firefox 3, so don't tell Demand Five in case they mess it up again!
Up until last Friday, I was watching Neighbours using Firefox 3. Yes, I know that's really sad! On Monday, I went to watch Neighbours and was greeted by a new Media Player window displaying:
"Your flash plug-in is out of date
Please download the latest version here"
No problemo, thought I. I updated my flash plug-in and went back to watch Neighbours, to be greeted by the new Media Player window displaying:
"Your flash plug-in is out of date
Please download the latest version here"
Uh-oh! I contacted the Demand Five Support Team informing them of my problem. I got the following reply:
"Greetings,
Thank you for contacting the Demand Five Support Team.
Please try to access with Internet Explorer..."
I stopped reading at that point as I don't use Internet Explorer. I e-mailed the Support Team informing them of that fact. I got the following reply:
"Greetings,
We apologize for the inconvenience, since our contents are related with DRM therefore our service is only compatible with Internet Explorer. All other browsers (e.g., Firefox, Opera, Safari, etc...) are not compatible at this time..."
They left out Google Chrome! So basically, Demand Five just alienated a large number of their users by making their site incompatible with every browser except Internet Explorer. Thanks a bunch! (That's an ironic thank you, for the benefit of foreign readers).
As I really wanted to watch Neighbours, I ran Internet Explorer, updated my flash plug-in and off I went. Monday's episode played O.K. but Tuesday's episode stopped after the 15 second Weight-Watcher's intro'. I e-mailed the Support Team informing them of that fact. I got the following reply:
"Greetings,
Please upgrade your DRM security at the following site:
http://go.microsoft.com/FWLink?LinkID=34506
The Demand Five Support Team
downloadsupport@five.tv
AM"
I clicked the link and pressed the Upgrade button. It didn't work. I e-mailed the Support Team informing them of that fact. I got the following reply:
"Greetings,
Thank you for contacting the Demand Five Support Team.
Please follow the instructions given below:
Please open Windows Media Player (WMP)
In the menu area at the top of the WMP window, click "Tools"
If "Tools" is not visible, Right-click on the upper bar area on WMP and a Menu-list will appear
In the list that appears, choose "Options"
In the window that opens, the "Player" tab will be the first tab displayed
Please ensure that both "Download codecs automatically" and "Connect to the Internet" are selected
Please select the "File Types" tab
Click "Select All", located below and to the right of the list
Click "Apply", located at the bottom of the "Options" window
Please select the "Network" tab
In the "Protocols for MMS URLs" section, un-check "RTSP/UDP" and "RTSP/TCP"
Now, re-check "RTSP/UDP" and "RTSP/TCP"
All three protocols should now be selected
Click "OK" at the bottom of the "Options" Window
Please close Windows Media Player."
It worked. I e-mailed the Support Team informing them of that fact and also asked them why Demand Five couldn't be as easy to use as BBC iPlayer. I didn't get a reply. I have posted the above information so that you too can watch Neighbours....everybody needs good Neighbours....
UPDATE: The Demand Five media player now works with Firefox 3 and Safari. This may mean that it now also works with Opera & Chrome.
According to the support page, it's still not compatible with Firefox 3, so don't tell Demand Five in case they mess it up again!
Labels:
BBC iPlayer,
Channel 5,
Demand Five,
DRM,
Firefox,
Internet Explorer,
Opera,
Safari,
Windows Media Player
Food Network CR
My Life In Food
Tune In:
Jan 31, 2009
6:30 PM ET/PT
"Fountain of Youth"
"My Life in Food follows two stories,a forty year-old athlete who believes his strict raw food diet is the secret weapon to out-run men half his age and a a couple who are part of a growing society that dramatically restricts their calories to half that of the average American."
Unfortunately I don't hav access to this because I don't believe we have it here in the UK... hope someone can get a DVR of it and post it up sometime.
Tune In:
Jan 31, 2009
6:30 PM ET/PT
"Fountain of Youth"
"My Life in Food follows two stories,a forty year-old athlete who believes his strict raw food diet is the secret weapon to out-run men half his age and a a couple who are part of a growing society that dramatically restricts their calories to half that of the average American."
Unfortunately I don't hav access to this because I don't believe we have it here in the UK... hope someone can get a DVR of it and post it up sometime.
The CR Way
Three videos I just got around to uploading for you, enjoy.
Vitamin K2 and Cranial Development
One of the things Dr. Weston Price noticed about healthy traditional cultures worldwide is their characteristically broad faces, broad dental arches and wide nostrils. Due to the breadth of their dental arches, they invariably had straight teeth and enough room for wisdom teeth. As soon as these same groups adopted white flour and sugar, the next generation to be born grew up with narrow faces, narrow dental arches, crowded teeth, pinched nostrils and a characteristic underdevelopment of the middle third of the face.
Here's an excerpt from Nutrition and Physical Degeneration, about traditional and modernized Swiss groups. Keep in mind these are Europeans we're talking about (although he found the same thing in all the races he studied):
The dietary transitions Price observed were typically from mineral- and vitamin-rich whole foods to refined modern foods, predominantly white flour and sugar. The villagers of the Loetschental valley obtained their fat-soluble vitamins from pastured dairy, which is particularly rich in vitamin K2 MK-4.
In a modern society like the U.S., most people exhibit signs of poor cranial development. How many people do you know with perfectly straight teeth who never required braces? How many people do you know whose wisdom teeth erupted normally?
The archaeological record shows that our hunter-gatherer ancestors generally didn't have crooked teeth. Humans evolved to have dental arches in proportion to their tooth size, like all animals. Take a look at these chompers. That skull is from an archaeological site in the Sahara desert that predates agriculture in the region. Those beautiful teeth are typical of paleolithic humans and modern hunter-gatherers. Crooked teeth and impacted wisdom teeth are only as old as agriculture. However, Price found that with care, certain traditional cultures were able to build well-formed skulls on an agricultural diet.
So was Price on to something, or was he just cherry picking individuals that supported his hypothesis? It turns out there's a developmental syndrome in the literature that might shed some light on this. It's called Binder's syndrome. Here's a description from a review paper about Binder's syndrome (emphasis mine):
Warfarin is rat poison. It kills rats by causing them to lose their ability to form blood clots, resulting in massive hemmorhage. It does this by depleting vitamin K, which is necessary for the proper functioning of blood clotting factors. It's used (in small doses) in humans to thin the blood as a treatment for abnormal blood clots. As it turns out, Binder's syndrome can be caused by a number of things that interfere with vitamin K metabolism. The sensitive period for humans is the first trimester. I think we're getting warmer...
Another name for Binder's syndrome is "warfarin embryopathy". There happens to be a rat model of it. Dr. Bill Webster's group at the University of Sydney injected rats daily with warfarin for up to 12 weeks, beginning on the day they were born (rats have a different developmental timeline than humans). They also administered large doses of vitamin K1 along with it. This is to ensure the rats continue to clot normally, rather than hemorrhaging. Another notable property of warfarin that I've mentioned before is its ability to inhibit the conversion of vitamin K1 to vitamin K2 MK-4. Here's what they had to say about the rats:
Here are a few quotes from a review paper by Dr. Webster's group. I have to post the whole abstract because it's a gem:
Here's an excerpt from Nutrition and Physical Degeneration, about traditional and modernized Swiss groups. Keep in mind these are Europeans we're talking about (although he found the same thing in all the races he studied):
The reader will scarcely believe it possible that such marked differences in facial form, in the shape of the dental arches, and in the health condition of the teeth as are to be noted when passing from the highly modernized lower valleys and plains country in Switzerland to the isolated high valleys can exist. Fig. 3 shows four girls with typically broad dental arches and regular arrangement of the teeth. They have been born and raised in the Loetschental Valley or other isolated valleys of Switzerland which provide the excellent nutrition that we have been reviewing.Price attributed this physical change to a lack of minerals and the fat-soluble vitamins necessary to make good use of them: vitamin A, vitamin D and what he called "activator X"-- now known to be vitamin K2 MK-4. The healthy cultures he studied all had an adequate source of vitamin K2, but many ate very little K1 (which comes mostly from vegetables). Inhabitants of the Loetschental valley ate green vegetables only in summer, due to the valley's harsh climate. The rest of the year, the diet was limited chiefly to whole grain sourdough rye bread and pastured dairy products.
Another change that is seen in passing from the isolated groups with their more nearly normal facial developments, to the groups of the lower valleys, is the marked irregularity of the teeth with narrowing of the arches and other facial features... While in the isolated groups not a single case of a typical mouth breather was found, many were seen among the children of the lower-plains group. The children studied were from ten to sixteen years of age.
The dietary transitions Price observed were typically from mineral- and vitamin-rich whole foods to refined modern foods, predominantly white flour and sugar. The villagers of the Loetschental valley obtained their fat-soluble vitamins from pastured dairy, which is particularly rich in vitamin K2 MK-4.
In a modern society like the U.S., most people exhibit signs of poor cranial development. How many people do you know with perfectly straight teeth who never required braces? How many people do you know whose wisdom teeth erupted normally?
The archaeological record shows that our hunter-gatherer ancestors generally didn't have crooked teeth. Humans evolved to have dental arches in proportion to their tooth size, like all animals. Take a look at these chompers. That skull is from an archaeological site in the Sahara desert that predates agriculture in the region. Those beautiful teeth are typical of paleolithic humans and modern hunter-gatherers. Crooked teeth and impacted wisdom teeth are only as old as agriculture. However, Price found that with care, certain traditional cultures were able to build well-formed skulls on an agricultural diet.
So was Price on to something, or was he just cherry picking individuals that supported his hypothesis? It turns out there's a developmental syndrome in the literature that might shed some light on this. It's called Binder's syndrome. Here's a description from a review paper about Binder's syndrome (emphasis mine):
The essential features of maxillo-nasal dysplasia were initially described by Noyes in 1939, although it was Binder who first defined it as a distinct clinical syndrome. He reported on three cases and recorded six specific characteristics:5Allow me to translate: in Binder's patients, the middle third of the face is underdeveloped, they have narrow dental arches and crowded teeth, small nostrils and abnormally small sinuses (sometimes resulting in mouth breathing). Sound familiar? So what causes Binder's syndrome? I'll give you a hint: it can be caused by prenatal exposure to warfarin (coumadin).Individuals with Binder's syndrome have a characteristic appearance that is easily recognizable.6 The mid-face profile is hypoplastic, the nose is flattened, the upper lip is convex with a broad philtrum, the nostrils are typically crescent or semi-lunar in shape due to the short collumela, and a deep fold or fossa occurs between the upper lip and the nose, resulting in an acute nasolabial angle.
- Arhinoid face.
- Abnormal position of nasal bones.
- Inter-maxillary hypoplasia with associated malocclusion.
- Reduced or absent anterior nasal spine.
- Atrophy of nasal mucosa.
- Absence of frontal sinus (not obligatory).
Warfarin is rat poison. It kills rats by causing them to lose their ability to form blood clots, resulting in massive hemmorhage. It does this by depleting vitamin K, which is necessary for the proper functioning of blood clotting factors. It's used (in small doses) in humans to thin the blood as a treatment for abnormal blood clots. As it turns out, Binder's syndrome can be caused by a number of things that interfere with vitamin K metabolism. The sensitive period for humans is the first trimester. I think we're getting warmer...
Another name for Binder's syndrome is "warfarin embryopathy". There happens to be a rat model of it. Dr. Bill Webster's group at the University of Sydney injected rats daily with warfarin for up to 12 weeks, beginning on the day they were born (rats have a different developmental timeline than humans). They also administered large doses of vitamin K1 along with it. This is to ensure the rats continue to clot normally, rather than hemorrhaging. Another notable property of warfarin that I've mentioned before is its ability to inhibit the conversion of vitamin K1 to vitamin K2 MK-4. Here's what they had to say about the rats:
The warfarin-treated rats developed a marked maxillonasal hypoplasia associated with a 11-13% reduction in the length of the nasal bones compared with controls... It is proposed that (1) the facial features of the human warfarin embryopathy are caused by reduced growth of the embryonic nasal septum, and (2) the septal growth retardation occurs because the warfarin-induced extrahepatic vitamin K deficiency prevents the normal formation of the vitamin K-dependent matrix gla protein in the embryo."Maxillonasal hypoplasia" means underdevelopment of the jaws and nasal region. Proper development of this region requires fully active matrix gla protein (MGP), which I've written about before in the context of vascular calcification. MGP requires vitamin K to activate it, and it seems to prefer K2 MK-4 to K1, at least in the vasculature. Administering K2 MK-4 along with warfarin prevents warfarin's ability to cause arterial calcification (thought to be an MGP-dependent mechanism), whereas administering K1 does not.
Here are a few quotes from a review paper by Dr. Webster's group. I have to post the whole abstract because it's a gem:
The normal vitamin K status of the human embryo appears to be close to deficiency [I would argue in most cases the embryo is actually deficient, as are most adults in industrial societies]. Maternal dietary deficiency or use of a number of therapeutic drugs during pregnancy, may result in frank vitamin K deficiency in the embryo. First trimester deficiency results in maxillonasal hypoplasia in the neonate with subsequent facial and orthodontic implications. A rat model of the vitamin K deficiency embryopathy shows that the facial dysmorphology is preceded by uncontrolled calcification in the normally uncalcified nasal septal cartilage, and decreased longitudinal growth of the cartilage, resulting in maxillonasal hypoplasia. The developing septal cartilage is normally rich in the vitamin K-dependent protein matrix gla protein (MGP). It is proposed that functional MGP is necessary to maintain growing cartilage in a non-calcified state. Developing teeth contain both MGP and a second vitamin K-dependent protein, bone gla protein (BGP). It has been postulated that these proteins have a functional role in tooth mineralization. As yet this function has not been established and abnormalities in tooth formation have not been observed under conditions where BGP and MGP should be formed in a non-functional form.Could vitamin K insufficiency be related to underdeveloped facial structure in industrialized cultures? Price felt that to ensure the proper development of their children, mothers should eat a diet rich in fat-soluble vitamins both before and during pregnancy. This makes sense in light of what we now know. There is a pool of vitamin K2 MK-4 in the organs that turns over very slowly, in addition to a pool in the blood that turns over rapidly. Entering pregnancy with a full store means a greater chance of having enough of the vitamin for the growing fetus. Healthy traditional cultures often fed special foods rich in fat-soluble vitamins to women of childbearing age and expectant mothers, thus ensuring beautiful and robust progeny.
Tuesday, January 27, 2009
The Protein-Sparing Modified Fast (PSMF)
As promised, I shall discuss the PSMF (I was going to have a whinge about the sudden loss of ability to watch "Neighbours" on Demand Five using Firefox, but I'll save that for another time if Demand Five Support don't fix the problem).
What's a PSMF?
A standard PSMF is ~1g of protein for every kg bodyweight a day plus lots of green leafy vegetables plus six to ten fish oil capsules a day plus vitamin & mineral supplements plus unlimited water AND NOTHING ELSE. You may find this quite literally hard to swallow! PSMF may also stand for Protein Strictly , Mother-F***er! A 100kg person (e.g. me) may get to eat ~400kcals from protein + ~100kcals from incidental carbohydrates & fats = ~500kcals a day. Hear that rumbling noise? It's my tummy! A well-known PSMF is Lyle McDonald's Rapid Fat Loss Handbook.
For more information, see http://forums.lylemcdonald.com/forumdisplay.php?f=7 and Is Rapid Fat Loss Right For You?
To make a PSMF easier to manage (but have a slower rate of weight loss), here are some modifications:-
1) Instead of six to ten fish oil capsules a day, stir ~25g of powdered linseeds into a large glass of drink and swallow the lot. Do this at breakfast-time. 25g of linseeds contains ~10g of fat (of which ~6g is Alpha-Linolenic Acid, an omega-3 fatty acid) which does the following:-
a) It stimulates the gall-bladder to empty, thus reducing the risk of gallstones.
b) It usually results in a bowel movement some time later. The ~10g of soluble fibre/fiber in the linseeds + accompanying fluid guarantees regularity.
c) It provides women (but not men) with all of the omega-3 fat they need each day.
Men need to eat either half a 213g tin of wild red salmon a day or six to ten fish oil capsules a day as their bodies don't produce enough DHA from Alpha-Linolenic Acid. See Extremely Limited Synthesis of Long Chain Polyunsaturates in Adults: Implications for their Dietary Essentiality and use as Supplements
2) Eat about 100g of protein a day. As meat, poultry & fish contains 20-25% protein, this means that you can eat ~1lb of meat, poultry & fish a day. 100g of protein a day is well within the capabilities of your liver and kidneys.
3) Eat about 44g of fat a day. This allows you to choose less lean cuts of meat & poultry and you can even eat the skin on chicken as long as you factor it into your total fat allowance. It also allows you to use vinaigrette salad dressings or a knob of butter or a dollop of real mayonnaise to make your vegetables taste nicer.
4) Eat about 50g of carbohydrate a day. This allows you to eat shed-loads of leafy green vegetables and also an onion. It also allows you to eat a portion of fruit e.g. an apple or a bowl of berries/cherries with Splenda & a dollop of whipped cream each day.
5) If you do any intense exercise (e.g. HIIT or resistance training with weights), eat an extra 50g of slow-release carbohydrates a couple of hours beforehand to fuel it.
6) Supplement with 5,000iu/day of Vitamin D3. Nowadays, many of us spend our lives mostly indoors and this causes many of us to become deficient in Vitamin D. Please read Vitamin D.
In conclusion:
100g of protein provides 400kcals, 44g of fat provides 400kcals and 50g of carbohydrate provides 200kcals, making a grand total of 1,000kcals a day. Hopefully, this will be enough to stop your tummy from rumbling. If you weigh over 100lbs but aren't losing weight on 1,000kcals a day, see your GP as you may have a thyroid problem.
I believe that the above diet tackles the problems of gallstones, constipation, dry skin, dry hair, depression and dietary deficiencies. You get to eat real food and quite a lot of it too, for a fairly rapid fat loss diet.
What's a PSMF?
A standard PSMF is ~1g of protein for every kg bodyweight a day plus lots of green leafy vegetables plus six to ten fish oil capsules a day plus vitamin & mineral supplements plus unlimited water AND NOTHING ELSE. You may find this quite literally hard to swallow! PSMF may also stand for Protein Strictly , Mother-F***er! A 100kg person (e.g. me) may get to eat ~400kcals from protein + ~100kcals from incidental carbohydrates & fats = ~500kcals a day. Hear that rumbling noise? It's my tummy! A well-known PSMF is Lyle McDonald's Rapid Fat Loss Handbook.
For more information, see http://forums.lylemcdonald.com/forumdisplay.php?f=7 and Is Rapid Fat Loss Right For You?
To make a PSMF easier to manage (but have a slower rate of weight loss), here are some modifications:-
1) Instead of six to ten fish oil capsules a day, stir ~25g of powdered linseeds into a large glass of drink and swallow the lot. Do this at breakfast-time. 25g of linseeds contains ~10g of fat (of which ~6g is Alpha-Linolenic Acid, an omega-3 fatty acid) which does the following:-
a) It stimulates the gall-bladder to empty, thus reducing the risk of gallstones.
b) It usually results in a bowel movement some time later. The ~10g of soluble fibre/fiber in the linseeds + accompanying fluid guarantees regularity.
c) It provides women (but not men) with all of the omega-3 fat they need each day.
Men need to eat either half a 213g tin of wild red salmon a day or six to ten fish oil capsules a day as their bodies don't produce enough DHA from Alpha-Linolenic Acid. See Extremely Limited Synthesis of Long Chain Polyunsaturates in Adults: Implications for their Dietary Essentiality and use as Supplements
2) Eat about 100g of protein a day. As meat, poultry & fish contains 20-25% protein, this means that you can eat ~1lb of meat, poultry & fish a day. 100g of protein a day is well within the capabilities of your liver and kidneys.
3) Eat about 44g of fat a day. This allows you to choose less lean cuts of meat & poultry and you can even eat the skin on chicken as long as you factor it into your total fat allowance. It also allows you to use vinaigrette salad dressings or a knob of butter or a dollop of real mayonnaise to make your vegetables taste nicer.
4) Eat about 50g of carbohydrate a day. This allows you to eat shed-loads of leafy green vegetables and also an onion. It also allows you to eat a portion of fruit e.g. an apple or a bowl of berries/cherries with Splenda & a dollop of whipped cream each day.
5) If you do any intense exercise (e.g. HIIT or resistance training with weights), eat an extra 50g of slow-release carbohydrates a couple of hours beforehand to fuel it.
6) Supplement with 5,000iu/day of Vitamin D3. Nowadays, many of us spend our lives mostly indoors and this causes many of us to become deficient in Vitamin D. Please read Vitamin D.
In conclusion:
100g of protein provides 400kcals, 44g of fat provides 400kcals and 50g of carbohydrate provides 200kcals, making a grand total of 1,000kcals a day. Hopefully, this will be enough to stop your tummy from rumbling. If you weigh over 100lbs but aren't losing weight on 1,000kcals a day, see your GP as you may have a thyroid problem.
I believe that the above diet tackles the problems of gallstones, constipation, dry skin, dry hair, depression and dietary deficiencies. You get to eat real food and quite a lot of it too, for a fairly rapid fat loss diet.
Labels:
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Monday, January 26, 2009
The Tokelau Island Migrant Study: The Final Word
Over the course of the last month, I've outlined some of the major findings of the Tokelau Island Migrant study. It's one of the most comprehensive studies I've found of a traditional culture transitioning to a modern diet and lifestyle. It traces the health of the inhabitants of the Pacific island Tokelau over time, as well as the health of Tokelauan migrants to New Zealand.
Unfortunately, the study began after the introduction of modern foods. We will never know for sure what Tokelauan health was like when their diet was completely traditional. To get some idea, we have to look at other traditional Pacific islanders such as the Kitavans.
What we can say is that an increase in the consumption of modern foods on Tokelau, chiefly white wheat flour and refined sugar, correlated with an increase in several non-communicable disorders, including overweight, diabetes and severe tooth decay. Further modernization as Tokelauans migrated to New Zealand corresponded with an increase in nearly every disorder measured, including heart disease, weight gain, diabetes, asthma and gout. These are all "diseases of civilization", which are not observed in hunter-gatherers and certain non-industrial populations throughout the world.
One of the most interesting things about Tokelauans is their extreme saturated fat intake, 40- 50% of calories. That's more than any other population I'm aware of. Yet Tokelauans appear to have a low incidence of heart attacks, lower than their New Zealand- dwelling relatives who eat half as much saturated fat. This should not be buried in the scientific literature; it should be common knowledge.
Overall, I believe the Tokelau Island Migrant study (among others) shows us that partially replacing nourishing traditional foods with modern foods such as processed wheat and sugar, is enough to cause a broad range of disorders not seen in hunter-gatherers but typical of modern societies. Changes in lifestyle between Tokelau and New Zealand may have also played a role.
The Tokelau Island Migrant Study: Background and Overview
The Tokelau Island Migrant Study: Dental Health
The Tokelau Island Migrant Study: Cholesterol and Cardiovascular Health
The Tokelau Island Migrant Study: Weight Gain
The Tokelau Island Migrant Study: Diabetes
The Tokelau Island Migrant Study: Asthma
The Tokelau Island Migrant Study: Gout
Unfortunately, the study began after the introduction of modern foods. We will never know for sure what Tokelauan health was like when their diet was completely traditional. To get some idea, we have to look at other traditional Pacific islanders such as the Kitavans.
What we can say is that an increase in the consumption of modern foods on Tokelau, chiefly white wheat flour and refined sugar, correlated with an increase in several non-communicable disorders, including overweight, diabetes and severe tooth decay. Further modernization as Tokelauans migrated to New Zealand corresponded with an increase in nearly every disorder measured, including heart disease, weight gain, diabetes, asthma and gout. These are all "diseases of civilization", which are not observed in hunter-gatherers and certain non-industrial populations throughout the world.
One of the most interesting things about Tokelauans is their extreme saturated fat intake, 40- 50% of calories. That's more than any other population I'm aware of. Yet Tokelauans appear to have a low incidence of heart attacks, lower than their New Zealand- dwelling relatives who eat half as much saturated fat. This should not be buried in the scientific literature; it should be common knowledge.
Overall, I believe the Tokelau Island Migrant study (among others) shows us that partially replacing nourishing traditional foods with modern foods such as processed wheat and sugar, is enough to cause a broad range of disorders not seen in hunter-gatherers but typical of modern societies. Changes in lifestyle between Tokelau and New Zealand may have also played a role.
The Tokelau Island Migrant Study: Background and Overview
The Tokelau Island Migrant Study: Dental Health
The Tokelau Island Migrant Study: Cholesterol and Cardiovascular Health
The Tokelau Island Migrant Study: Weight Gain
The Tokelau Island Migrant Study: Diabetes
The Tokelau Island Migrant Study: Asthma
The Tokelau Island Migrant Study: Gout
60 Minutes Red Wine, Rhesus Monkey, CRONies
Although the CR part is not all that good in my opinion, looks like the editor never did a good job here because there was very little on human CRers, or the amazing health results. However I think the Rhesus Monkey CR part makes up for it...
Make sure you listen out for the statistics on the mortality of the rhesus monkeys :) Loooooking goood for CR!
Go to this LINK or press play on the video below
Make sure you listen out for the statistics on the mortality of the rhesus monkeys :) Loooooking goood for CR!
Go to this LINK or press play on the video below
Sunday, January 25, 2009
Very Low Calorie Diets (VLCDs)
A well-known VLCD is The Cambridge Diet. Lighter Life is another VLCD.
The VLCD is, as its name suggests, very low in Calories and is aimed at morbidly obese people i.e. people who have a Body Mass Index (BMI) of over 40. Such people are at a very high risk of dropping dead of a heart attack and they are also at a high risk of complications caused by high blood pressure, high blood glucose, high blood triglycerides, high blood cholesterol, high blood LDL-c, low blood HDL-c and high blood uric acid. In addition, morbidly obese people have breathing problems e.g. sleep apnoea and they are also at a high risk of dying while under anaesthetic if they need to be operated on. Such people need to lose weight rapidly. However, people who are overweight (BMI 25-29.9) or obese (BMI 30-39.9) or who are just unhappy with their bodies should not embark on a VLCD as the risks outweigh the benefits.
1) VLCDs result in rapid weight loss. You may think that this is a good thing, but rapid weight loss brings with it problems.
a) Excessive loss of muscle. This is more of a problem for women who, because they have naturally-low testosterone levels, have great difficulty regaining any lost muscle.
b) High risk of developing Gallstones. Rapid weight loss results in an increase in the concentration of cholesterol in bile. This increases the risk factor for gallstones, something that women have a higher risk factor for than men. There's an acronym FFFF for people who are at a high risk of developing gallstones. It stands for Female, Forty, Fat, Fair. What makes the situation even worse is that VLCDs are very low in fat. The gallbladder is a muscular bag which stores bile. When dietary fat is eaten, this stimulates the secretion of cholecystokinin, which then stimulates contraction of the gallbladder muscle, which expels bile from inside the gallbladder into the duodenum. The lower the fat content of a meal, the less the gallbladder contracts and gallbladder stasis can result with only 2g of fat per meal. See The role of gallbladder emptying in gallstone formation during diet-induced rapid weight loss. The problem with this study is that the two groups of subjects were not eating the same number of calories. See Gallbladder motility and gallstone formation in obese patients following very low calorie diets. Use it (fat) to lose it (well) and Similarity in gallstone formation from 900 kcal/day diets containing 16 g vs 30 g of daily fat: evidence that fat restriction is not the main culprit of cholelithiasis during rapid weight reduction.
In these studies, both groups were on the same calorie intake. In the second study, 17% of the low-fat group developed gallstones whereas only 11.2% of the higher-fat group developed gallstones.
The other problem with a very low fat intake is EFA deficiency. Essential Fatty Acids are called that for a reason....they are essential for us to live. Dry skin & hair are common on VLCDs. The small amount of fat that there is in a VLCD almost certainly contains mostly omega-6 polyunsaturates. A lack of omega-3 EFAs can adversely affect mental function. Depression is common on VLCDs. See Omega-3 fatty acids and major depression: A primer for the mental health professional. Another problem with very low fat intakes is a lack of fat-soluble vitamins, particularly Vitamins D3 and K. See Vitamin D and Vitamin K.
2) VLCDs contain excessive amounts of sugars. This has two problems.
a) Unstable blood glucose levels. See Blood Glucose, Insulin & Diabetes. You don't notice peaks in blood glucose. However, dips cause severe hunger pangs and, in some people, neurosis. See Hypoglycemia & Neurosis.
b) Carbohydrates fill glycogen stores. As glycogen stores become filled, fat-burning decreases. When glycogen stores become full, fat-burning falls to zero. Fat-burning increases again as glycogen stores deplete between meals - but you feel hungry.
Sedentary people's bodies don't burn much carbohydrate. See Everyone is Different. At rest, on average a fasted person derives ~65% of energy from fats and ~35% from carbohydrate, although there are extremes of 93% fat-burning to 20% fat-burning. Even if someone who has full glycogen stores derives 100% of their energy at rest from carbohydrate, as they are only burning ~1kcal/minute at rest, their body is only burning 0.25g of carbohydrate/minute. So why feed someone carbohydrate when their body doesn't need it?
c) People with excess belly fat almost certainly have The Metabolic Syndrome. This causes various problems including high serum triglycerides (TGs). Eating carbohydrate that isn't burned and can't be stored raises TGs. I know about this as I have blood test results which showed TGs increasing with increasing carbohydrate intake. High TGs are bad news for your arteries. See Cholesterol And Coronary Heart Disease.
3) VLCDs don't contain enough protein. Protein supplies Amino Acids (AAs) to the body. These are used to preserve muscle mass. AAs can also be used to generate blood glucose in the liver by a process called Gluconeogenesis (GNG), which makes the consumption of carbohydrates redundant for most sedentary people.
4) VLCDs don't contain enough fibre/fiber. Constipation is common on VLCDs.
In my next Blog post, I will discuss a Rapid Fat Loss alternative to the VLCD that overcomes all of the above problems and is therefore much safer and more pleasant to be on. See The Protein-Sparing Modified Fast (PSMF)
For a discussion of VLCDs, see What do you think of Very Low Energy Diets?
The VLCD is, as its name suggests, very low in Calories and is aimed at morbidly obese people i.e. people who have a Body Mass Index (BMI) of over 40. Such people are at a very high risk of dropping dead of a heart attack and they are also at a high risk of complications caused by high blood pressure, high blood glucose, high blood triglycerides, high blood cholesterol, high blood LDL-c, low blood HDL-c and high blood uric acid. In addition, morbidly obese people have breathing problems e.g. sleep apnoea and they are also at a high risk of dying while under anaesthetic if they need to be operated on. Such people need to lose weight rapidly. However, people who are overweight (BMI 25-29.9) or obese (BMI 30-39.9) or who are just unhappy with their bodies should not embark on a VLCD as the risks outweigh the benefits.
1) VLCDs result in rapid weight loss. You may think that this is a good thing, but rapid weight loss brings with it problems.
a) Excessive loss of muscle. This is more of a problem for women who, because they have naturally-low testosterone levels, have great difficulty regaining any lost muscle.
b) High risk of developing Gallstones. Rapid weight loss results in an increase in the concentration of cholesterol in bile. This increases the risk factor for gallstones, something that women have a higher risk factor for than men. There's an acronym FFFF for people who are at a high risk of developing gallstones. It stands for Female, Forty, Fat, Fair. What makes the situation even worse is that VLCDs are very low in fat. The gallbladder is a muscular bag which stores bile. When dietary fat is eaten, this stimulates the secretion of cholecystokinin, which then stimulates contraction of the gallbladder muscle, which expels bile from inside the gallbladder into the duodenum. The lower the fat content of a meal, the less the gallbladder contracts and gallbladder stasis can result with only 2g of fat per meal. See The role of gallbladder emptying in gallstone formation during diet-induced rapid weight loss. The problem with this study is that the two groups of subjects were not eating the same number of calories. See Gallbladder motility and gallstone formation in obese patients following very low calorie diets. Use it (fat) to lose it (well) and Similarity in gallstone formation from 900 kcal/day diets containing 16 g vs 30 g of daily fat: evidence that fat restriction is not the main culprit of cholelithiasis during rapid weight reduction.
In these studies, both groups were on the same calorie intake. In the second study, 17% of the low-fat group developed gallstones whereas only 11.2% of the higher-fat group developed gallstones.
The other problem with a very low fat intake is EFA deficiency. Essential Fatty Acids are called that for a reason....they are essential for us to live. Dry skin & hair are common on VLCDs. The small amount of fat that there is in a VLCD almost certainly contains mostly omega-6 polyunsaturates. A lack of omega-3 EFAs can adversely affect mental function. Depression is common on VLCDs. See Omega-3 fatty acids and major depression: A primer for the mental health professional. Another problem with very low fat intakes is a lack of fat-soluble vitamins, particularly Vitamins D3 and K. See Vitamin D and Vitamin K.
2) VLCDs contain excessive amounts of sugars. This has two problems.
a) Unstable blood glucose levels. See Blood Glucose, Insulin & Diabetes. You don't notice peaks in blood glucose. However, dips cause severe hunger pangs and, in some people, neurosis. See Hypoglycemia & Neurosis.
b) Carbohydrates fill glycogen stores. As glycogen stores become filled, fat-burning decreases. When glycogen stores become full, fat-burning falls to zero. Fat-burning increases again as glycogen stores deplete between meals - but you feel hungry.
Sedentary people's bodies don't burn much carbohydrate. See Everyone is Different. At rest, on average a fasted person derives ~65% of energy from fats and ~35% from carbohydrate, although there are extremes of 93% fat-burning to 20% fat-burning. Even if someone who has full glycogen stores derives 100% of their energy at rest from carbohydrate, as they are only burning ~1kcal/minute at rest, their body is only burning 0.25g of carbohydrate/minute. So why feed someone carbohydrate when their body doesn't need it?
c) People with excess belly fat almost certainly have The Metabolic Syndrome. This causes various problems including high serum triglycerides (TGs). Eating carbohydrate that isn't burned and can't be stored raises TGs. I know about this as I have blood test results which showed TGs increasing with increasing carbohydrate intake. High TGs are bad news for your arteries. See Cholesterol And Coronary Heart Disease.
3) VLCDs don't contain enough protein. Protein supplies Amino Acids (AAs) to the body. These are used to preserve muscle mass. AAs can also be used to generate blood glucose in the liver by a process called Gluconeogenesis (GNG), which makes the consumption of carbohydrates redundant for most sedentary people.
4) VLCDs don't contain enough fibre/fiber. Constipation is common on VLCDs.
In my next Blog post, I will discuss a Rapid Fat Loss alternative to the VLCD that overcomes all of the above problems and is therefore much safer and more pleasant to be on. See The Protein-Sparing Modified Fast (PSMF)
For a discussion of VLCDs, see What do you think of Very Low Energy Diets?
Labels:
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Depression,
Diabetes,
EFAs,
Gallstones,
Hypoglycemia,
Lighter Life,
Metabolic Syndrome,
Neurosis,
The Cambridge Diet,
Type 2 Diabetes,
Very Low Calorie Diets,
Vitamin D,
Vitamin D3,
Vitamin K,
VLCDs
The Tokelau Island Migrant Study: Gout
Gout is a disorder in which uric acid crystals form in the joints, causing intense pain. The body forms uric acid as a by-product of purine metabolism. Purines are a building block of DNA, among other things. Uric acid is normally excreted into the urine, hence the name.
On Tokelau between 1971 and 1982, gout prevalence fell slightly. In migrants to New Zealand, gout prevalence began at the same level as on Tokelau but increased rapidly over the same time period. Here are the prevalence data for men, from Migration and Health in a Small Society: the Case of Tokelau (I don't have data for women):
This paper found that the age-standardized risk of developing gout was 9 times higher in New Zealand than on Tokelau for men, and 2.7 times higher for women.
The Tokelau Island Migrant Study: Background and Overview
The Tokelau Island Migrant Study: Dental Health
The Tokelau Island Migrant Study: Cholesterol and Cardiovascular Health
The Tokelau Island Migrant Study: Weight Gain
The Tokelau Island Migrant Study: Diabetes
The Tokelau Island Migrant Study: Asthma
On Tokelau between 1971 and 1982, gout prevalence fell slightly. In migrants to New Zealand, gout prevalence began at the same level as on Tokelau but increased rapidly over the same time period. Here are the prevalence data for men, from Migration and Health in a Small Society: the Case of Tokelau (I don't have data for women):
This paper found that the age-standardized risk of developing gout was 9 times higher in New Zealand than on Tokelau for men, and 2.7 times higher for women.
The Tokelau Island Migrant Study: Background and Overview
The Tokelau Island Migrant Study: Dental Health
The Tokelau Island Migrant Study: Cholesterol and Cardiovascular Health
The Tokelau Island Migrant Study: Weight Gain
The Tokelau Island Migrant Study: Diabetes
The Tokelau Island Migrant Study: Asthma
Cranberry-Apricot Muffins (Gluten, Dairy and Sugar Free)
By the time you read this post, Dear Reader, I'll be walking Venice Beach. And sighing a long slow sigh. At last. A long overdue getaway- the first vacation since our last visit to the City of Angels- way back in the fall of 2007. Which, considering the roller coaster ride of a year we've all had- personally and collectively- feels like a virtual lifetime ago.
I'll be away for a week. But don't worry. You know I couldn't leave you on your own without a new recipe to tempt you into the kitchen. So I stirred up a new batter for you- some tender breakfast muffins. A fresh combination of tart cranberries and golden apricot preserves in a flour base that features buckwheat, sorghum and quinoa flours- yielding a higher protein profile than your average gluten-free muffin. And, as a bonus to those of you eschewing sugar cane, this recipe has no added sugar. (I didn't add nuts this time, but if you like a little nutty crunch in your muffins these would be lovely with chopped pecans, or toasted almonds.)
Read more + get the recipe >>
CR only work in obese?
I was going to discuss this here but I'll just direct you to a thread at imminst, which also has links to the papers from these authors that claim CR has no benefit in non obese humans because of their failure to extend a mouse which is a bit screwed up.
FORUM: Mouse Study Suggests CR only works in obese?
I really recommend you read that thread, and ignore these big headline that make ridiculous claims. Long Term CRer Michael has some very good points so make sure you read that post.
FORUM: Mouse Study Suggests CR only works in obese?
I really recommend you read that thread, and ignore these big headline that make ridiculous claims. Long Term CRer Michael has some very good points so make sure you read that post.
Friday, January 23, 2009
Why exercise may or may not help you to lose weight.
Exercise. I hate it. I'm about as active as a Brazilian 2-toed Sloth. The problem that I have with exercise is that it makes me feel really hungry and I end up eating more Calories than I burned by exercising. This is due to the effect of AMPk on my brain.
The thing about exercise in moderation is that it's good for health and fitness. The problem is that some people think that lots more is better. That ain't necessarily so. The 38 year old lady sitting near me at karaoke last night used to run a lot when she was at school. She now needs a replacement knee joint due to damaged cartilage and she had her knee wired up to a TENS machine.
Too much early morning exercise can also make you ill, by raising serum Cortisol levels. Excessive Cortisol is immunosuppressive. See Early morning exercise could make you ill. Excessive Cortisol also causes water retention, so people who over-exercise can gain (water) weight. Excessive Cortisol can also cause muscle loss, thin skin and osteoporosis.
Starving yourself and over-exercising just makes things even worse. From WHY is the combination of high intensity and/or long duration activity a mistake when calories are being severely restricted?
"Water retention: cortisol binds to the mineralocorticoid receptor (the receptor involved in water retention, well one of them). And although cortisol has 1/100th of the effect on water balance of the primary hormones (aldosterone and a couple of others), since there is like 8000 times as much of it, it can cause a major effect." and
"Excessive cortisol, especially chronic elevations cause other problems not the least of which is leptin resistance. Which only magnifies the drop in leptin from dieting. This could be another mechanism behind the greater drop in metabolic rate for the study I mentioned above."
So, what's the best thing to do for maximum fat loss with minimal muscle loss? A mixture of high-intensity exercise (resistance training with weights, or sprinting) and medium-intensity exercise (jogging) is better than just medium-intensity exercise. See Resistance Weight Training With Endurance Training Enhances Fat Loss, Impact of Exercise Intensity on Body Fatness and Skeletal Muscle Metabolism and HIIT & Run.
One theory to try and explain the improved fat loss is that Calories are burned after the high-intensity exercise is finished. However, the Excess Post-exercise Oxygen Consumption (EPOC) only amounts to about 35kcals so it isn't significant. What is significant is the appetite reduction produced by high-intensity exercise, the opposite effect from low and medium-intensity exercise.
The thing about exercise in moderation is that it's good for health and fitness. The problem is that some people think that lots more is better. That ain't necessarily so. The 38 year old lady sitting near me at karaoke last night used to run a lot when she was at school. She now needs a replacement knee joint due to damaged cartilage and she had her knee wired up to a TENS machine.
Too much early morning exercise can also make you ill, by raising serum Cortisol levels. Excessive Cortisol is immunosuppressive. See Early morning exercise could make you ill. Excessive Cortisol also causes water retention, so people who over-exercise can gain (water) weight. Excessive Cortisol can also cause muscle loss, thin skin and osteoporosis.
Starving yourself and over-exercising just makes things even worse. From WHY is the combination of high intensity and/or long duration activity a mistake when calories are being severely restricted?
"Water retention: cortisol binds to the mineralocorticoid receptor (the receptor involved in water retention, well one of them). And although cortisol has 1/100th of the effect on water balance of the primary hormones (aldosterone and a couple of others), since there is like 8000 times as much of it, it can cause a major effect." and
"Excessive cortisol, especially chronic elevations cause other problems not the least of which is leptin resistance. Which only magnifies the drop in leptin from dieting. This could be another mechanism behind the greater drop in metabolic rate for the study I mentioned above."
So, what's the best thing to do for maximum fat loss with minimal muscle loss? A mixture of high-intensity exercise (resistance training with weights, or sprinting) and medium-intensity exercise (jogging) is better than just medium-intensity exercise. See Resistance Weight Training With Endurance Training Enhances Fat Loss, Impact of Exercise Intensity on Body Fatness and Skeletal Muscle Metabolism and HIIT & Run.
One theory to try and explain the improved fat loss is that Calories are burned after the high-intensity exercise is finished. However, the Excess Post-exercise Oxygen Consumption (EPOC) only amounts to about 35kcals so it isn't significant. What is significant is the appetite reduction produced by high-intensity exercise, the opposite effect from low and medium-intensity exercise.
Labels:
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AMPk,
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EPOC,
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Leptin,
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Water retention
Thursday, January 22, 2009
The Tokelau Island Migrant Study: Asthma
Asthma may be another "disease of civilization", uncommon in non-industrial cultures. Between 1980 and 2001, its prevalence more than doubled in American children 17 years and younger. The trend is showing no sign of slowing down (CDC NHANES surveys).
The age-standardized asthma prevalence in Tokelauan migrants to New Zealand age 15 and older, was 2 - 6 times higher than in non-migrants from 1976 to 1982, depending on gender and year. The highest prevalence was in New Zealand migrant women in 1976, at 6.8%. The lowest was in Tokelauan men in 1976 at 1.1%.
A skeptic might suggest it's because these adults grew up around certain types of pollen or other antigens, and were exposed to new ones later in life. However, even migrant children in the 0-4 age group, who were most likely born in NZ, had more asthma than on Tokelau.
What could contribute to the increased asthma prevalence upon modernization? I'm not particularly knowledgeable about the mechanisms of asthma, but it seems likely to involve a chronic over-activation of the immune system ("inflammation").
The Tokelau Island Migrant Study: Background and Overview
The Tokelau Island Migrant Study: Dental Health
The Tokelau Island Migrant Study: Cholesterol and Cardiovascular Health
The Tokelau Island Migrant Study: Weight Gain
The Tokelau Island Migrant Study: Diabetes
The Tokelau Island Migrant Study data in this post come from the book Migration and Health in a Small Society: The Case of Tokelau.
Thanks to the EPA and Wikipedia for the graph image (public domain).
The age-standardized asthma prevalence in Tokelauan migrants to New Zealand age 15 and older, was 2 - 6 times higher than in non-migrants from 1976 to 1982, depending on gender and year. The highest prevalence was in New Zealand migrant women in 1976, at 6.8%. The lowest was in Tokelauan men in 1976 at 1.1%.
A skeptic might suggest it's because these adults grew up around certain types of pollen or other antigens, and were exposed to new ones later in life. However, even migrant children in the 0-4 age group, who were most likely born in NZ, had more asthma than on Tokelau.
What could contribute to the increased asthma prevalence upon modernization? I'm not particularly knowledgeable about the mechanisms of asthma, but it seems likely to involve a chronic over-activation of the immune system ("inflammation").
The Tokelau Island Migrant Study: Background and Overview
The Tokelau Island Migrant Study: Dental Health
The Tokelau Island Migrant Study: Cholesterol and Cardiovascular Health
The Tokelau Island Migrant Study: Weight Gain
The Tokelau Island Migrant Study: Diabetes
The Tokelau Island Migrant Study data in this post come from the book Migration and Health in a Small Society: The Case of Tokelau.
Thanks to the EPA and Wikipedia for the graph image (public domain).
Wednesday, January 21, 2009
Savory Vegetable Pancakes
I love savory pancakes- shredded veggies add so much flavor. |
Savory or sweet? That is my question today- not to indulge in the duality of either/or and divide the pancake universe into opposing sensibilities or anything, but if push comes to shove (and I'm four square against shoving, honestly) the naked truth is I'm more of a savory, salty, golden crunchy pancake lover than a soft, sweet and sticky with maple syrup craver.
Maybe it's because I love pancakes for supper.
Read more + get the recipe >>
Labels:
gluten free pancakes,
gluten free savory pancakes,
gluten-free vegetarian,
latkes,
vegetable
Tuesday, January 20, 2009
Magnesium: Just as important as Calcium.
Suffering from anger/aggression/anxiety? Can't sleep properly? Suffering from night cramps, restless legs, menstrual cramps, muscle spasms? You're probably deficient in magnesium. See A case of oesophageal spasm, and the ‘unproven’ treatment that helped it and Around the Web; and Menstrual Cramp Remedy.
After Vitamin D and Omega-3 fats, magnesium is the third thing that people are most likely to be deficient in. Processed foods are low in magnesium. Diets low in vegetables are low in magnesium. For a list of the 999 richest sources of magnesium per 100g serving, see http://nutritiondata.self.com/foods-000120000000000000000-w.html. Too much calcium can result in a relative magnesium deficiency.
An optimum intake of magnesium is approximately 50% of your calcium intake. Other sources of magnesium are Milk of Magnesia (hydroxide) and Epsom Salts (sulphate heptahydrate). Excessive intake of magnesium salts acts as an osmotic laxative and gives you loose bowels, but it takes quite a lot to do this. Half a level teaspoonful (~2.5g) of Epsom Salts gives you ~250mg of magnesium and is as cheap as chips (200g costs £1.25 at Boots).
Magnesium is also available as a dietary supplement. Magnesium oxide (Magnesia) isn't as well-absorbed as Citrate/Amino Acid Chelate forms, so take extra if using oxide. See Magnesium bioavailability from magnesium citrate and magnesium oxide. Magnesium can be absorbed through the skin, so adding Epsom Salts or Magnesium Chloride to your bathwater is another option.
After Vitamin D and Omega-3 fats, magnesium is the third thing that people are most likely to be deficient in. Processed foods are low in magnesium. Diets low in vegetables are low in magnesium. For a list of the 999 richest sources of magnesium per 100g serving, see http://nutritiondata.self.com/foods-000120000000000000000-w.html. Too much calcium can result in a relative magnesium deficiency.
An optimum intake of magnesium is approximately 50% of your calcium intake. Other sources of magnesium are Milk of Magnesia (hydroxide) and Epsom Salts (sulphate heptahydrate). Excessive intake of magnesium salts acts as an osmotic laxative and gives you loose bowels, but it takes quite a lot to do this. Half a level teaspoonful (~2.5g) of Epsom Salts gives you ~250mg of magnesium and is as cheap as chips (200g costs £1.25 at Boots).
Magnesium is also available as a dietary supplement. Magnesium oxide (Magnesia) isn't as well-absorbed as Citrate/Amino Acid Chelate forms, so take extra if using oxide. See Magnesium bioavailability from magnesium citrate and magnesium oxide. Magnesium can be absorbed through the skin, so adding Epsom Salts or Magnesium Chloride to your bathwater is another option.
Labels:
Aggression,
Anxiety,
Calcium,
Constipation,
Cramps,
Epsom Salts,
Insomnia,
Magnesium,
Menstrual cramps,
Milk of Magnesia,
Muscle spasms,
Night cramps,
Period pains,
Restless legs,
Spasms
The Tokelau Island Migrant Study: Diabetes
This post will be short and sweet. Diabetes is a disease of civilization. As Tokelauans adopted Western industrial foods, their diabetes prevalence increased. At any given time point, age-standardized diabetes prevalence was higher in migrants to New Zealand than those who remained on Tokelau:
This is not a difference in diagnosis. Tokelauans were examined for diabetes by the same group of physicians, using the same criteria. It's also not a difference in average age, sice the numbers are age-standardized. On Tokelau, diabetes prevalence doubled in a decade. Migrants to New Zealand in 1981 had roughly three times the prevalence of diabetes that Tokelauans did in 1971. I can only imagine the prevalence is even higher in 2008.
We don't know what the prevalence was in Tokelauans when their diet was completely traditional, but I would expect it to be low like other traditional Pacific island societies. I'm looking at a table right now of age-standardized diabetes prevalence on 11 different Pacific islands. There is quite a bit of variation, but the pattern is clear: the more modernized, the higher the diabetes rate. In several cases, the table has placed two values side-by-side: one value for rural inhabitants of an island, and another for urban inhabitants of the same island. In every case, the prevalence of diabetes is higher in the urban group. In some cases, the difference is as large as four-fold.
The lowest value goes to the New Caledonians of Touho, who are also considered the least modernized on the table (although even their diet is not completely traditional). Men have an age-standardized diabetes prevalence of 1.8%, women 1.4%. At the other extreme are the Micronesians of Nauru, affluent due to phosphate resources, who have a prevalence of 33.4% for men and 32.1% for women. They subsist mostly on imported food and are extremely obese.
The same patterns can be seen in Africa, the Arctic and probably everywhere that has adopted processed Western foods. White rice alone (compared with the combination of wheat flour and sugar) does not seem to have this effect.
The data in this post are from the book Migration and Health in a Small Society: the Case of Tokelau.
The Tokelau Island Migrant Study: Background and Overview
The Tokelau Island Migrant Study: Dental Health
The Tokelau Island Migrant Study: Cholesterol and Cardiovascular Health
The Tokelau Island Migrant Study: Weight Gain
This is not a difference in diagnosis. Tokelauans were examined for diabetes by the same group of physicians, using the same criteria. It's also not a difference in average age, sice the numbers are age-standardized. On Tokelau, diabetes prevalence doubled in a decade. Migrants to New Zealand in 1981 had roughly three times the prevalence of diabetes that Tokelauans did in 1971. I can only imagine the prevalence is even higher in 2008.
We don't know what the prevalence was in Tokelauans when their diet was completely traditional, but I would expect it to be low like other traditional Pacific island societies. I'm looking at a table right now of age-standardized diabetes prevalence on 11 different Pacific islands. There is quite a bit of variation, but the pattern is clear: the more modernized, the higher the diabetes rate. In several cases, the table has placed two values side-by-side: one value for rural inhabitants of an island, and another for urban inhabitants of the same island. In every case, the prevalence of diabetes is higher in the urban group. In some cases, the difference is as large as four-fold.
The lowest value goes to the New Caledonians of Touho, who are also considered the least modernized on the table (although even their diet is not completely traditional). Men have an age-standardized diabetes prevalence of 1.8%, women 1.4%. At the other extreme are the Micronesians of Nauru, affluent due to phosphate resources, who have a prevalence of 33.4% for men and 32.1% for women. They subsist mostly on imported food and are extremely obese.
The same patterns can be seen in Africa, the Arctic and probably everywhere that has adopted processed Western foods. White rice alone (compared with the combination of wheat flour and sugar) does not seem to have this effect.
The data in this post are from the book Migration and Health in a Small Society: the Case of Tokelau.
The Tokelau Island Migrant Study: Background and Overview
The Tokelau Island Migrant Study: Dental Health
The Tokelau Island Migrant Study: Cholesterol and Cardiovascular Health
The Tokelau Island Migrant Study: Weight Gain
Labels:
diabetes,
disease,
diseases of civilization,
Tokelau
Comparison shot
I find some picture I had for a while and thought it would make a good comparison shot of what I was like when I was 18 ande how I am now at 24. The shot is from a diferent angle but still quite good. I can spot one huge difference, can you? CLICK ON THE PICTURE TO ENLARGE
BEFORE AND AFTER PHOTO
BEFORE AND AFTER PHOTO
Sunday, January 18, 2009
Green Smothies
I have been eating less amount of raw food lately and probably more cooked food. I don't think this is the best way to eat healthy so I decided to get out the blender and try out green smoothie, which I can get 4 servings from and lasts me 2 days. I have one in the morning and one in the evening. When I cosume I chew it rather than just drinking because chewing gets the saliva going. My second one contained;
Bananas 200g
Apples 180g
Kale 250g
Spinach 250g
Blueberries 100g
Pineapple 150g
Water 350ml (upto 500ml water is good and might be better for some, just adjust serving size).
Very simple, very delicious and packed full of good nutrition.
I'm still working out the exact ingredients as I want to get all nutrients a bit higher. I think lower amount of bananas is perferable because of less sugar (but using more bananas does taste a bit better). In saying that 100g of banana a day isn't really a lot and the the 2nd smoothie was 146k/cal per serving (370g) which is great. It's an excellent way to start of the day I find, and if eating a big salad isn't your thing in the morning then you're getting lots of greens and some fruit right away. Enzymes, vitamins and minerals, fatty acids, fibres, Energy! It makes you feel wonderful and light. Some people are put off by the colour (I actually like it) but it tastes amazing! If you have any recommendation or would like to share you smoothie recipe please comment below ;)
You can find plenty of green smoothie demonstration on youtube. Enjoy.
Now all I need to start saving for is a Vita-Mix Blender! :D
PER SERVING;
General (8%)
===========================================
Energy | 146.5 kcal 6%
Protein | 4.9 g 3%
Carbs | 34.7 g 13%
Fiber | 6.1 g 16%
Fat | 1.1 g 1%
Water | 326.6 g 9%
Vitamins (37%)
===========================================
Vitamin A | 15562.2 IU 519%
Folate | 159.0 µg 40%
B1 (Thiamine) | 0.2 mg 15%
B2 (Riboflavin) | 0.3 mg 21%
B3 (Niacin) | 1.7 mg 11%
B5 (Pantothenic Acid)| 0.4 mg 8%
B6 (Pyridoxine) | 0.5 mg 42%
B12 (Cyanocobalamin) | 0.0 µg 0%
Vitamin C | 119.3 mg 133%
Vitamin D | 0.0 IU 0%
Vitamin E | 1.5 mg 10%
Vitamin K | 818.8 µg 682%
Minerals (24%)
===========================================
Calcium | 160.4 mg 16%
Copper | 0.4 mg 42%
Iron | 3.1 mg 39%
Magnesium | 93.3 mg 23%
Manganese | 1.6 mg 71%
Phosphorus | 87.6 mg 13%
Potassium | 915.4 mg 19%
Selenium | 1.8 µg 3%
Sodium | 81.3 mg 5%
Zinc | 0.8 mg 7%
Lipids (4%)
===========================================
Saturated | 0.2 g 1%
Omega-3 | 0.2 g 15%
Omega-6 | 0.2 g 1%
Cholesterol | 0.0 mg 0%
Bananas 200g
Apples 180g
Kale 250g
Spinach 250g
Blueberries 100g
Pineapple 150g
Water 350ml (upto 500ml water is good and might be better for some, just adjust serving size).
Very simple, very delicious and packed full of good nutrition.
I'm still working out the exact ingredients as I want to get all nutrients a bit higher. I think lower amount of bananas is perferable because of less sugar (but using more bananas does taste a bit better). In saying that 100g of banana a day isn't really a lot and the the 2nd smoothie was 146k/cal per serving (370g) which is great. It's an excellent way to start of the day I find, and if eating a big salad isn't your thing in the morning then you're getting lots of greens and some fruit right away. Enzymes, vitamins and minerals, fatty acids, fibres, Energy! It makes you feel wonderful and light. Some people are put off by the colour (I actually like it) but it tastes amazing! If you have any recommendation or would like to share you smoothie recipe please comment below ;)
You can find plenty of green smoothie demonstration on youtube. Enjoy.
Now all I need to start saving for is a Vita-Mix Blender! :D
PER SERVING;
General (8%)
===========================================
Energy | 146.5 kcal 6%
Protein | 4.9 g 3%
Carbs | 34.7 g 13%
Fiber | 6.1 g 16%
Fat | 1.1 g 1%
Water | 326.6 g 9%
Vitamins (37%)
===========================================
Vitamin A | 15562.2 IU 519%
Folate | 159.0 µg 40%
B1 (Thiamine) | 0.2 mg 15%
B2 (Riboflavin) | 0.3 mg 21%
B3 (Niacin) | 1.7 mg 11%
B5 (Pantothenic Acid)| 0.4 mg 8%
B6 (Pyridoxine) | 0.5 mg 42%
B12 (Cyanocobalamin) | 0.0 µg 0%
Vitamin C | 119.3 mg 133%
Vitamin D | 0.0 IU 0%
Vitamin E | 1.5 mg 10%
Vitamin K | 818.8 µg 682%
Minerals (24%)
===========================================
Calcium | 160.4 mg 16%
Copper | 0.4 mg 42%
Iron | 3.1 mg 39%
Magnesium | 93.3 mg 23%
Manganese | 1.6 mg 71%
Phosphorus | 87.6 mg 13%
Potassium | 915.4 mg 19%
Selenium | 1.8 µg 3%
Sodium | 81.3 mg 5%
Zinc | 0.8 mg 7%
Lipids (4%)
===========================================
Saturated | 0.2 g 1%
Omega-3 | 0.2 g 15%
Omega-6 | 0.2 g 1%
Cholesterol | 0.0 mg 0%
Saturday, January 17, 2009
The Tokelau Island Migrant Study: Weight Gain
Between 1968 and 1982, Tokelauans in nearly all age groups gained weight, roughly 5 kilograms (11 pounds) on average. They also became slightly taller, but not enough to offset the gain in weight. By 1980-82, migrants to New Zealand had become especially heavy, with all age groups weighing more than non-migrants by about 5 kg (11 lb) on average, and 10 kg (22 lb) more than Tokelauans did in 1968.
The body mass index (BMI) is a rough estimate of fat mass (although it can be confounded by muscle mass), and is the weight in kilograms divided by the square of the height in meters [BMI = weight / (height^2)]. A BMI of 25 to 30 is considered overweight; 30 and over is considered obese.
The graphs I'm about to present require some explanation. The data in each graph were collected from the same individuals over time (15-69 years old). That means some weight gain is expected, as this population normally gains weight into middle age (then loses weight). What's interesting to note is the difference in the rate of weight change between migrants and non-migrants. The first two data points in 1968 are baseline, and compare non-migrants with "pre-migrants" still living on Tokelau. The second two data points in 1981-82 compare the same individual migrants in New Zealand with the same non-migrants.
Unless they all decided to become body builders, migrants to New Zealand gained more fat mass than Tokelauans between 1968 and 1982. The rate of weight gain in New Zealand was more than twice as fast for men and more than 50% faster for women than on Tokelau.
Why did Tokelauans and especially migrants to New Zealand gain weight? Probably because they had greater access to a wide variety of calorie-dense, palatable foods of modern commerce. The introduction of wheat and sugar, at the expense of coconut and traditional carbohydrate sources, was the main change to the Tokelauan diet during this time period. See this post for a graph.
Finally, there's the question of exercise. Did a change in energy expenditure contribute to weight gain? The study didn't collect data on exercise during the time period in question, so all we have are anecdotes. During this time, men living on Tokelau progressively adopted outboard motors for their fishing boats, replacing the traditional sails and oars. Their energy expenditure probably decreased.
But what about women? Tokelauan women traditionally perform household tasks such as weaving mats and preparing food. Their energy expenditure probably didn't change much over the same time period. Since both men and women on Tokelau gained weight, it would be hard to argue that exercise was a dominant factor.
How about migrants to New Zealand? Here's a quote from Migration and Health in a Small Society: the Case of Tokelau:
The body mass index (BMI) is a rough estimate of fat mass (although it can be confounded by muscle mass), and is the weight in kilograms divided by the square of the height in meters [BMI = weight / (height^2)]. A BMI of 25 to 30 is considered overweight; 30 and over is considered obese.
The graphs I'm about to present require some explanation. The data in each graph were collected from the same individuals over time (15-69 years old). That means some weight gain is expected, as this population normally gains weight into middle age (then loses weight). What's interesting to note is the difference in the rate of weight change between migrants and non-migrants. The first two data points in 1968 are baseline, and compare non-migrants with "pre-migrants" still living on Tokelau. The second two data points in 1981-82 compare the same individual migrants in New Zealand with the same non-migrants.
Unless they all decided to become body builders, migrants to New Zealand gained more fat mass than Tokelauans between 1968 and 1982. The rate of weight gain in New Zealand was more than twice as fast for men and more than 50% faster for women than on Tokelau.
Why did Tokelauans and especially migrants to New Zealand gain weight? Probably because they had greater access to a wide variety of calorie-dense, palatable foods of modern commerce. The introduction of wheat and sugar, at the expense of coconut and traditional carbohydrate sources, was the main change to the Tokelauan diet during this time period. See this post for a graph.
Finally, there's the question of exercise. Did a change in energy expenditure contribute to weight gain? The study didn't collect data on exercise during the time period in question, so all we have are anecdotes. During this time, men living on Tokelau progressively adopted outboard motors for their fishing boats, replacing the traditional sails and oars. Their energy expenditure probably decreased.
But what about women? Tokelauan women traditionally perform household tasks such as weaving mats and preparing food. Their energy expenditure probably didn't change much over the same time period. Since both men and women on Tokelau gained weight, it would be hard to argue that exercise was a dominant factor.
How about migrants to New Zealand? Here's a quote from Migration and Health in a Small Society: the Case of Tokelau:
Overall it is our belief that most of the migrants expend greater energy in their work than is currently the case in Tokelau.Exercise doesn't appear to have been the main factor, although the data don't allow us to be totally confident about this.
Labels:
diet,
exercise,
native diet,
overweight,
Tokelau
Friday, January 16, 2009
I've got a lovely bunch of coconuts!
Coconut Oil is the No. 1 most stable oil for cooking at high temperatures.
What’s in coconut oil?
According to http://www.manitobaharvest.com/nutrition/index.asp?itemID=183 , coconut oil is ~91% saturated fatty acids, ~7% monounsaturated fatty acids, ~2% omega-6 fatty acids and zero omega-3 fatty acids.
According to McCance and Widdowson's “The Composition of Foods”, the fatty acid composition of coconut oil is as follows:-
Name(:0=sat, :1=mono, :2=poly, n6=omega-6) Quantity (%)
Caprylic Acid (C8:0)___________________________7.5
Capric Acid (C10:0)____________________________7.1
Lauric Acid (C12:0)___________________________47.7
Myristic Acid (C14:0)_________________________15.8
Palmitic Acid (C16:0)__________________________9.0
Stearic Acid (C18:0)___________________________2.4
Arachidic Acid (C20:0)_________________________1.0
Palmitoleic Acid (C16:1)_______________________0.4
Oleic Acid (C18:1)_____________________________6.6
Linoleic Acid (C18:2 n6)_______________________1.8
Won’t all that saturated fat give me a heart attack?
Whether or not you get coronary heart disease depends on your whole diet. According to http://www.ajcn.org/cgi/reprint/34/8/1552.pdf , Pukapukans got 26% (male) to 30% (female) of their total Calories from saturated fats. Tokelauans got 47% (male) to 49% (female) of their total Calories from saturated fats. Tokelauans had total serum cholesterol 35-40mg/dL (0.9- 1.03mmol/L) higher than Pukapukans.
As Tokelauans were getting about seven times more energy from saturated fats than the 7% that current healthy eating guidelines recommend, they must have been dropping like flies from coronary heart disease or strokes, right? Wrong! To quote:-
“Vascular disease is uncommon is both populations and there is no evidence of the high saturated fat intake having a harmful effect in these populations.”
How come? Well, if you look at the rest of the Tokelauans’ diet, you’ll see virtually no refined sugar or cereal products. Basically, they weren’t eating any junk. When Tokelauans migrated to New Zealand, their sat fat intake fell to ~41% of total calories but as they were eating more refined carbs & sugar, their lipid profile got worse.
What are the benefits of coconut oil?
Medium-chain fatty acids are metabolised rapidly without passing through the liver and they provide a quick source of energy for muscles. There is some evidence that medium-chain fatty acids stimulate the thyroid gland to secrete more T4 & T3 which can be an aid when cutting. There is also some evidence that Lauric Acid has anti-bacterial & anti-viral properties. Coconut oil is also good for the skin when rubbed in.
Where can I buy coconut oil?
Don’t buy cheap coconut oil. It’s almost certainly Refined, Bleached & Deodorised which detracts from its health benefits. The best coconut oils are Organic Virgin Oils. Some good on-line sources are
http://www.fresh-coconut.com/ ,
Coconut Oil UK and
http://www.revital.co.uk/product_search.cfm?searchString="Nutiva+Organic+Extra+Virgin+Coconut+Oil"
In addition, check-out Rosso's Blog on Saturated oils.
What’s in coconut oil?
According to http://www.manitobaharvest.com/nutrition/index.asp?itemID=183 , coconut oil is ~91% saturated fatty acids, ~7% monounsaturated fatty acids, ~2% omega-6 fatty acids and zero omega-3 fatty acids.
According to McCance and Widdowson's “The Composition of Foods”, the fatty acid composition of coconut oil is as follows:-
Name(:0=sat, :1=mono, :2=poly, n6=omega-6) Quantity (%)
Caprylic Acid (C8:0)___________________________7.5
Capric Acid (C10:0)____________________________7.1
Lauric Acid (C12:0)___________________________47.7
Myristic Acid (C14:0)_________________________15.8
Palmitic Acid (C16:0)__________________________9.0
Stearic Acid (C18:0)___________________________2.4
Arachidic Acid (C20:0)_________________________1.0
Palmitoleic Acid (C16:1)_______________________0.4
Oleic Acid (C18:1)_____________________________6.6
Linoleic Acid (C18:2 n6)_______________________1.8
Won’t all that saturated fat give me a heart attack?
Whether or not you get coronary heart disease depends on your whole diet. According to http://www.ajcn.org/cgi/reprint/34/8/1552.pdf , Pukapukans got 26% (male) to 30% (female) of their total Calories from saturated fats. Tokelauans got 47% (male) to 49% (female) of their total Calories from saturated fats. Tokelauans had total serum cholesterol 35-40mg/dL (0.9- 1.03mmol/L) higher than Pukapukans.
As Tokelauans were getting about seven times more energy from saturated fats than the 7% that current healthy eating guidelines recommend, they must have been dropping like flies from coronary heart disease or strokes, right? Wrong! To quote:-
“Vascular disease is uncommon is both populations and there is no evidence of the high saturated fat intake having a harmful effect in these populations.”
How come? Well, if you look at the rest of the Tokelauans’ diet, you’ll see virtually no refined sugar or cereal products. Basically, they weren’t eating any junk. When Tokelauans migrated to New Zealand, their sat fat intake fell to ~41% of total calories but as they were eating more refined carbs & sugar, their lipid profile got worse.
What are the benefits of coconut oil?
Medium-chain fatty acids are metabolised rapidly without passing through the liver and they provide a quick source of energy for muscles. There is some evidence that medium-chain fatty acids stimulate the thyroid gland to secrete more T4 & T3 which can be an aid when cutting. There is also some evidence that Lauric Acid has anti-bacterial & anti-viral properties. Coconut oil is also good for the skin when rubbed in.
Where can I buy coconut oil?
Don’t buy cheap coconut oil. It’s almost certainly Refined, Bleached & Deodorised which detracts from its health benefits. The best coconut oils are Organic Virgin Oils. Some good on-line sources are
http://www.fresh-coconut.com/ ,
Coconut Oil UK and
http://www.revital.co.uk/product_search.cfm?searchString="Nutiva+Organic+Extra+Virgin+Coconut+Oil"
In addition, check-out Rosso's Blog on Saturated oils.
Labels:
Coconut,
Coconut oil,
Lauric Acid,
McCance and Widdowson,
Monounsaturated fatty acids,
Omega-3,
Omega-6,
Pukapukans,
Saturated fatty acids,
Serum cholesterol,
The Composition of Foods,
Tokelauans
Thursday, January 15, 2009
Gluten-Free Banana-Blueberry Muffin Cake
My partner in culinary crime- the infamous and ever-up-for-baking husband Steve- informed me our bananas were mucho ripe. In fact, to be completely accurate, he poked his head into my cozy little studio the other day and declared, Our bananas are ripe. I should bake something.
I looked up from my iMac and murmured Hmmm, in assent.
But what, exactly?
Ripe bananas were calling. Begging to be a part of some grander life affirming tastebud tingling scheme. But do readers really need another banana cake recipe? I pondered, slurping cold coffee with vanilla hemp milk. Which reminds me. I should share this.
The Tokelau Island Migrant Study: Cholesterol and Cardiovascular Health
Let's get right to the meat of this study. It's relevant to the hypothesis that saturated fat is a cause of cardiovascular disease. Tokelauans traditionally obtained 40-50% of their calories from saturated fat, in the form of coconut meat. That's more than any other group I'm aware of.
So are the Tokelauans dropping like flies of cardiovascular disease? I don't have access to the best data of all: actual heart attack incidence data. But we do have some telltale markers. In 1971-1982, researchers collected data from Tokelau and Tokelauan migrants to New Zealand on cholesterol levels, blood pressure and electrocardiogram (ECG) readings.
The Tokelauan diet, as I've described in detail in previous posts, is traditionally based on coconut, fish, starchy tubers and fruit. By 1982, their diet also contained a significant amount of imported flour and sugar. Migrants to New Zealand had a much more varied diet that was also more typically Western: more carbohydrate, coming chiefly from wheat, sugar and potatoes; more processed sweet foods and drinks; more red meat; more vegetables; more dairy and eggs. Sugar intake was 13 percent of calories, compared to 8 percent on Tokelau. Saturated fat intake in NZ was half of what it was on Tokelau, while total fat intake was similar. Polyunsaturated fat intake was higher in NZ, 4% as opposed to 2% in Tokelau. I don't have data to back this up, but I think it's likely that the n-6:n-3 ratio increased upon migration.
Blood pressure did not change significantly over time in Tokelau from 1971 to 1982, if anything it actually declined slightly. It was consistently higher in NZ than in Tokelau at all timepoints. Men were roughly three times more likely to be hypertensive in NZ than on Tokelau at all timepoints (4.0% vs. 12.0% in the early 1970s). Women were about twice as likely to be hypertensive (8.1% vs. 15.0%).
On to cholesterol. Total cholesterol in male Tokelauans was a bit lower on average than in New Zealand, but neither was particularly elevated (182 vs. 199 mg/dL). LDL was also a bit higher in NZ males (119 vs. 132 mg/dL). Triglycerides were lower in Tokelauan men than in NZ (80 vs. 114 mg/dL). There were no differences in total cholesterol, LDL cholesterol or triglycerides between Tokelauan and NZ women. It's interesting that serum lipids don't correspond at all to saturated fat intake.
But does it cause heart attacks? The best data I have from this study are ECG readings. These use electrodes to monitor the electrical activity of the heart. There are certain ECG patterns that suggest that a person has had a heart attack (Minnesota codes 1-1 and 1-2). The data I am going to present here are all age-standardized, meaning they are comparing between groups of the same age. On Tokelau in 1982, 0.0% of men 40-69 years old showed ECG readings that indicated a probable past heart attack. In NZ in 1980-81, 1.0% of men 40-69 years old showed the same ECG readings. In Tecumseh U.S.A. in 1965, 3.5% of men 40-69 years old showed the same ECG pattern. I don't have data for women.
These data don't prove that no one ever has a heart attack on Tokelau. Tokelauans do have heart attacks sometimes, and they also have strokes (at least in modern times). But they do allow us to compare in quantitative terms between genetically similar people living in two different environments.
This is consistent with what has been observed on Kitava and other traditional Pacific island cultures: a vanishingly small incidence of cardiovascular disease while they retain their traditional diet and lifestyle (and sometimes even when some processed Western food has been introduced). When diets and lifestyles become modern, there is invariably a rise in the incidence of chronic disease.
These data raise serious questions about the role of saturated fat in cardiovascular disease. Tokelau underlines the fact that a non-industrial diet and lifestyle may be a more significant protective factor than the quality of ingested fat.
Unless otherwise noted, the data in this post are from the book Migration and Health in a Small Society: the Case of Tokelau.
So are the Tokelauans dropping like flies of cardiovascular disease? I don't have access to the best data of all: actual heart attack incidence data. But we do have some telltale markers. In 1971-1982, researchers collected data from Tokelau and Tokelauan migrants to New Zealand on cholesterol levels, blood pressure and electrocardiogram (ECG) readings.
The Tokelauan diet, as I've described in detail in previous posts, is traditionally based on coconut, fish, starchy tubers and fruit. By 1982, their diet also contained a significant amount of imported flour and sugar. Migrants to New Zealand had a much more varied diet that was also more typically Western: more carbohydrate, coming chiefly from wheat, sugar and potatoes; more processed sweet foods and drinks; more red meat; more vegetables; more dairy and eggs. Sugar intake was 13 percent of calories, compared to 8 percent on Tokelau. Saturated fat intake in NZ was half of what it was on Tokelau, while total fat intake was similar. Polyunsaturated fat intake was higher in NZ, 4% as opposed to 2% in Tokelau. I don't have data to back this up, but I think it's likely that the n-6:n-3 ratio increased upon migration.
Blood pressure did not change significantly over time in Tokelau from 1971 to 1982, if anything it actually declined slightly. It was consistently higher in NZ than in Tokelau at all timepoints. Men were roughly three times more likely to be hypertensive in NZ than on Tokelau at all timepoints (4.0% vs. 12.0% in the early 1970s). Women were about twice as likely to be hypertensive (8.1% vs. 15.0%).
On to cholesterol. Total cholesterol in male Tokelauans was a bit lower on average than in New Zealand, but neither was particularly elevated (182 vs. 199 mg/dL). LDL was also a bit higher in NZ males (119 vs. 132 mg/dL). Triglycerides were lower in Tokelauan men than in NZ (80 vs. 114 mg/dL). There were no differences in total cholesterol, LDL cholesterol or triglycerides between Tokelauan and NZ women. It's interesting that serum lipids don't correspond at all to saturated fat intake.
But does it cause heart attacks? The best data I have from this study are ECG readings. These use electrodes to monitor the electrical activity of the heart. There are certain ECG patterns that suggest that a person has had a heart attack (Minnesota codes 1-1 and 1-2). The data I am going to present here are all age-standardized, meaning they are comparing between groups of the same age. On Tokelau in 1982, 0.0% of men 40-69 years old showed ECG readings that indicated a probable past heart attack. In NZ in 1980-81, 1.0% of men 40-69 years old showed the same ECG readings. In Tecumseh U.S.A. in 1965, 3.5% of men 40-69 years old showed the same ECG pattern. I don't have data for women.
These data don't prove that no one ever has a heart attack on Tokelau. Tokelauans do have heart attacks sometimes, and they also have strokes (at least in modern times). But they do allow us to compare in quantitative terms between genetically similar people living in two different environments.
This is consistent with what has been observed on Kitava and other traditional Pacific island cultures: a vanishingly small incidence of cardiovascular disease while they retain their traditional diet and lifestyle (and sometimes even when some processed Western food has been introduced). When diets and lifestyles become modern, there is invariably a rise in the incidence of chronic disease.
These data raise serious questions about the role of saturated fat in cardiovascular disease. Tokelau underlines the fact that a non-industrial diet and lifestyle may be a more significant protective factor than the quality of ingested fat.
Unless otherwise noted, the data in this post are from the book Migration and Health in a Small Society: the Case of Tokelau.
Labels:
Cardiovascular disease,
diet,
disease,
fats,
hypertension,
native diet,
Tokelau
Tuesday, January 13, 2009
As sure as Eggs is Eggs.....
...is grammatically incorrect! Rumour has it that it's a corruption of "As sure as x = x". But anyway....
Eggs are very nutritious and should be eaten freely as part of a healthy diet.
Click http://www.nutritiondata.com/facts/dairy-and-egg-products/111/2 and set the serving size to 1 large (50g) to see what nutrients there are in a raw whole egg.
Click http://www.nutritiondata.com/facts/dairy-and-egg-products/112/2 and set the serving size to 1 large (33g) to see what nutrients there are in a raw egg white.
Click http://www.nutritiondata.com/facts/dairy-and-egg-products/113/2 and set serving size to 1 large (17g) to see what nutrients there are in a raw egg yolk.
The amino acid scores for whole egg , white and yolk are 136, 145 and 146 respectively. Eggs are a good source of complete proteins. This is because the yolk and white need to contain everything necessary for a growing chick embryo.
Can I drink eggs raw to save time?There are three potential problems with this:
1) Salmonella poisoning. Unless you’re pretty sure of the hens that the eggs came from, there is a risk of poisoning from raw eggs. This doesn’t apply to pasteurised eggs from suppliers like http://www.eggnation.co.uk/ for example.
2) Poor absorption of egg white protein. According to http://jn.nutrition.org/cgi/content/full/128/10/1716 , only 51% of raw egg white protein is absorbed during digestion compared to 91% for cooked egg white protein. According to http://ajpgi.physiology.org/cgi/content/full/277/5/G935 , the figures are 65% and 94% respectively. The second study used 200g of white and one yolk. I don’t believe that there is a problem with the absorption of raw egg yolk, though problem 1 still remains. Pasteurised egg white protein is well absorbed.
3) Poor biotin absorption. Raw egg white contains a glycoprotein called avidin which binds to biotin (Vit. B7) in the yolk and prevents its absorption. Cooking or pasteurisation denatures (changes the 3-D structure of) the avidin and renders it harmless.
What about all that cholesterol in egg yolks?
Current “Healthy Eating” guidelines state that we should eat no more than 3 egg yolks/whole eggs per week. This is based on the erroneous assumption that dietary cholesterol always increases serum cholesterol and that this is always a bad thing. According to http://www.ajcn.org/cgi/reprint/32/5/1051.pdf , adding or not adding 500mg of dietary cholesterol from two large eggs per day made no significant difference to serum cholesterol or triglycerides in 116 healthy male subjects. Some went up and some went down. Eddie Vos at http://www.health-heart.org/cholesterol.htm reckons that you’d have to eat 20 whole eggs per day to get as much dietary cholesterol as the liver produces each day (5g). Egg yolks do contain some fat and this should be factored into your total diet. If you happen to have the genes for familial hypercholesterolaemia, then you need to keep a close eye on dietary cholesterol intake. See also Eat Whole Eggs All Day and Throw Your Statins Away? 375x Increased Dietary Cholesterol Intake From Eggs Reduces Visceral Fat & Promotes Healthy Cholesterol Metabolism.
There is a problem with modern eggs though, and it’s caused by the food that’s fed to the hens. Grains contain about 50 times more Linoleic acid (omega-6) than Alpha-Linolenic acid (omega-3) and this raises the omega-6:omega-3 ratio of the eggs that the hens lay. Hens eating a natural diet of bugs, grubs and vegetation lay eggs with a 1:1 ratio of omega-6:omega-3, but grain-fed hens lay eggs with an omega-6:omega-3 ratio of >10:1. A high omega-6:omega-3 ratio in the diet is associated with increased risk factors for heart disease, cancer and Insulin Resistance (pre-type 2 diabetes). Therefore, if large numbers of cheapo eggs are eaten, it’s advisable to eat other foods that are rich in omega-3 fats.
Eggs are very nutritious and should be eaten freely as part of a healthy diet.
Click http://www.nutritiondata.com/facts/dairy-and-egg-products/111/2 and set the serving size to 1 large (50g) to see what nutrients there are in a raw whole egg.
Click http://www.nutritiondata.com/facts/dairy-and-egg-products/112/2 and set the serving size to 1 large (33g) to see what nutrients there are in a raw egg white.
Click http://www.nutritiondata.com/facts/dairy-and-egg-products/113/2 and set serving size to 1 large (17g) to see what nutrients there are in a raw egg yolk.
The amino acid scores for whole egg , white and yolk are 136, 145 and 146 respectively. Eggs are a good source of complete proteins. This is because the yolk and white need to contain everything necessary for a growing chick embryo.
Can I drink eggs raw to save time?There are three potential problems with this:
1) Salmonella poisoning. Unless you’re pretty sure of the hens that the eggs came from, there is a risk of poisoning from raw eggs. This doesn’t apply to pasteurised eggs from suppliers like http://www.eggnation.co.uk/ for example.
2) Poor absorption of egg white protein. According to http://jn.nutrition.org/cgi/content/full/128/10/1716 , only 51% of raw egg white protein is absorbed during digestion compared to 91% for cooked egg white protein. According to http://ajpgi.physiology.org/cgi/content/full/277/5/G935 , the figures are 65% and 94% respectively. The second study used 200g of white and one yolk. I don’t believe that there is a problem with the absorption of raw egg yolk, though problem 1 still remains. Pasteurised egg white protein is well absorbed.
3) Poor biotin absorption. Raw egg white contains a glycoprotein called avidin which binds to biotin (Vit. B7) in the yolk and prevents its absorption. Cooking or pasteurisation denatures (changes the 3-D structure of) the avidin and renders it harmless.
What about all that cholesterol in egg yolks?
Current “Healthy Eating” guidelines state that we should eat no more than 3 egg yolks/whole eggs per week. This is based on the erroneous assumption that dietary cholesterol always increases serum cholesterol and that this is always a bad thing. According to http://www.ajcn.org/cgi/reprint/32/5/1051.pdf , adding or not adding 500mg of dietary cholesterol from two large eggs per day made no significant difference to serum cholesterol or triglycerides in 116 healthy male subjects. Some went up and some went down. Eddie Vos at http://www.health-heart.org/cholesterol.htm reckons that you’d have to eat 20 whole eggs per day to get as much dietary cholesterol as the liver produces each day (5g). Egg yolks do contain some fat and this should be factored into your total diet. If you happen to have the genes for familial hypercholesterolaemia, then you need to keep a close eye on dietary cholesterol intake. See also Eat Whole Eggs All Day and Throw Your Statins Away? 375x Increased Dietary Cholesterol Intake From Eggs Reduces Visceral Fat & Promotes Healthy Cholesterol Metabolism.
There is a problem with modern eggs though, and it’s caused by the food that’s fed to the hens. Grains contain about 50 times more Linoleic acid (omega-6) than Alpha-Linolenic acid (omega-3) and this raises the omega-6:omega-3 ratio of the eggs that the hens lay. Hens eating a natural diet of bugs, grubs and vegetation lay eggs with a 1:1 ratio of omega-6:omega-3, but grain-fed hens lay eggs with an omega-6:omega-3 ratio of >10:1. A high omega-6:omega-3 ratio in the diet is associated with increased risk factors for heart disease, cancer and Insulin Resistance (pre-type 2 diabetes). Therefore, if large numbers of cheapo eggs are eaten, it’s advisable to eat other foods that are rich in omega-3 fats.
Labels:
Alpha-linolenic acid,
Avidin,
Biotin,
Cholesterol,
Eggs,
Familial hypercholesterolaemia,
Linoleic acid,
Omega-3,
Omega-6,
Proteins,
Vitamin B7
Sunday, January 11, 2009
Linseeds/Flaxseeds & Flaxseed oil.
"Where flax is eaten...health abounds!" - Mahatma Ghandi.
These little seeds pack a quadruple-whammy of protein, omega-3 essential fatty acids (EFAs), soluble fibre/fiber, and minerals, vitamins and co-factors.
What's in flaxseeds and flaxseed oil?
Click http://www.nutritiondata.com/facts/nut-and-seed-products/3163/2 and set serving size to 100g to see what nutrients there are in flaxseeds.
Click http://www.nutritiondata.com/facts/fats-and-oils/7554/2 and set serving size to 100g to see what nutrients there are in flaxseed oil.
How do I eat flaxseeds and flaxseed oil?
Flaxseeds have a fibrous seed coat which swells-up when wet and passes through our guts undigested. To get the benefit of the protein, omega-3 essential fatty acids and minerals in flaxseeds, the seeds need to be crushed, cracked, chopped-up, sliced-up or ground-up using a coffee grinder, adjustable pepper grinder or most simply, a blender with a sharp blade. The resulting powder can be mixed with liquids or sprinkled on foods, though extra fluid must be drunk as the soluble fibre/fiber absorbs lots of water. Although whole flaxseeds will keep fresh at room temperature, once powdered, it's advisable to keep them in a cool dark place to minimise oxidation of any exposed fat*. Flaxseed oil must be kept refrigerated with the cap screwed on the bottle at all times after opening and it must never be used for cooking. It's O.K. sprinkled over hot food as long as the food is eaten shortly afterwards. Oxidised flaxseed oil tastes bitter and has lost any health benefits it had when fresh, so it should be discarded or used to varnish something or thin down putty. Unoxidised flaxseed oil has a nutty taste or it may taste slightly like tea. Ground-up flaxseeds hardly taste of anything.
*NEW! Milled flaxseed stability information.
How much flaxseeds and flaxseed oil do I need to eat each day?
Men are much poorer converters of alpha-linolenic acid (the omega-3 fatty acid in flaxseeds) into the longer-chain omega-3 fatty acids than women. See Eicosapentaenoic and docosapentaenoic acids are the principal products of α-linolenic acid metabolism in young men , Conversion of α-linolenic acid to eicosapentaenoic, docosapentaenoic and docosahexaenoic acids in young women and Extremely Limited Synthesis of Long Chain Polyunsaturates in Adults: Implications for Their Dietary Essentiality and Use as Supplements. Therefore, vegan men should eat ~50g of ground flaxseeds a day and women should eat ~25g a day. The daily amount of flaxseed oil for men is ~20g and the daily amount for women is ~10g. Vegan men should also supplement with vegan DHA.
Where can I buy flaxseeds and flaxseed oil?
Flaxseeds/linseeds come in different colours. The cheapest linseeds are brown/bronze ones which are often sold as bird seed in pet shops, but they can also be found in small independent health food shops. There are also golden linseeds, which is the type most often found in supermarkets. Linusit and Granovita are two well-known brands. Granovita organic flaxseed oil is a good brand and it comes in dark bottles to keep the light out as light causes photo-oxidation of omega-3 fatty acids.
These little seeds pack a quadruple-whammy of protein, omega-3 essential fatty acids (EFAs), soluble fibre/fiber, and minerals, vitamins and co-factors.
What's in flaxseeds and flaxseed oil?
Click http://www.nutritiondata.com/facts/nut-and-seed-products/3163/2 and set serving size to 100g to see what nutrients there are in flaxseeds.
Click http://www.nutritiondata.com/facts/fats-and-oils/7554/2 and set serving size to 100g to see what nutrients there are in flaxseed oil.
How do I eat flaxseeds and flaxseed oil?
Flaxseeds have a fibrous seed coat which swells-up when wet and passes through our guts undigested. To get the benefit of the protein, omega-3 essential fatty acids and minerals in flaxseeds, the seeds need to be crushed, cracked, chopped-up, sliced-up or ground-up using a coffee grinder, adjustable pepper grinder or most simply, a blender with a sharp blade. The resulting powder can be mixed with liquids or sprinkled on foods, though extra fluid must be drunk as the soluble fibre/fiber absorbs lots of water. Although whole flaxseeds will keep fresh at room temperature, once powdered, it's advisable to keep them in a cool dark place to minimise oxidation of any exposed fat*. Flaxseed oil must be kept refrigerated with the cap screwed on the bottle at all times after opening and it must never be used for cooking. It's O.K. sprinkled over hot food as long as the food is eaten shortly afterwards. Oxidised flaxseed oil tastes bitter and has lost any health benefits it had when fresh, so it should be discarded or used to varnish something or thin down putty. Unoxidised flaxseed oil has a nutty taste or it may taste slightly like tea. Ground-up flaxseeds hardly taste of anything.
*NEW! Milled flaxseed stability information.
How much flaxseeds and flaxseed oil do I need to eat each day?
Men are much poorer converters of alpha-linolenic acid (the omega-3 fatty acid in flaxseeds) into the longer-chain omega-3 fatty acids than women. See Eicosapentaenoic and docosapentaenoic acids are the principal products of α-linolenic acid metabolism in young men , Conversion of α-linolenic acid to eicosapentaenoic, docosapentaenoic and docosahexaenoic acids in young women and Extremely Limited Synthesis of Long Chain Polyunsaturates in Adults: Implications for Their Dietary Essentiality and Use as Supplements. Therefore, vegan men should eat ~50g of ground flaxseeds a day and women should eat ~25g a day. The daily amount of flaxseed oil for men is ~20g and the daily amount for women is ~10g. Vegan men should also supplement with vegan DHA.
Where can I buy flaxseeds and flaxseed oil?
Flaxseeds/linseeds come in different colours. The cheapest linseeds are brown/bronze ones which are often sold as bird seed in pet shops, but they can also be found in small independent health food shops. There are also golden linseeds, which is the type most often found in supermarkets. Linusit and Granovita are two well-known brands. Granovita organic flaxseed oil is a good brand and it comes in dark bottles to keep the light out as light causes photo-oxidation of omega-3 fatty acids.
Labels:
Alpha-linolenic acid,
EFAs,
Essential fatty acids,
Flaxseed oil,
Flaxseeds,
Linseeds,
Minerals,
Omega-3,
Photo-oxidation,
Protein,
Soluble fibre,
Vegan DHA,
Vitamins
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